Vaccines are causing an unprecedented number of mutations creating superbugs and potent viruses and bacteria that may eventually threaten future generations and humanity itself. Evidence continues to mount from the scientific community who now admit that certain vaccines are in-fact causing both viral and bacterial mutations. Ironically, the same researchers assert that “better” vaccines are needed to offset the rise in persistent mutations.
Life-threatening pathogens are capable of evolving rapidly and developing genetic decoys that serve to disguise them from even the most powerful drugs. University of Oxford researcher Rory Bowden found that pathogens switch genetic material with other bacteria, but predominantly for the part of the genome responsible for making the cell coating, which is the area targeted by vaccines.
Former post-doctoral researcher of the Center for Infectious Disease Dynamics, Grainne Long found that vaccination led to a 40-fold enhancement of B. parapertussis colonization in the lungs. His data suggested that the vaccine may be contributing to the observed rise in whooping cough incidence over the last decade by promoting B. parapertussis infection.
An acellular whooping cough vaccine actually enhances the colonization of Bordetella parapertussis in mice; pointing towards a rise in B. parapertussis incidence resulting from acellular vaccination, which may have contributed to the observed increase in whooping cough over the last decade.
Despite widespread vaccination, whooping cough incidence is on the rise worldwide, making it a disease virtually immune to vaccines.
Dangerous new strains of whooping cough bacteria are now evading Australia’s vaccine against the disease and entrenching a four-year epidemic that could soon spread overseas, Sydney scientists have found in research that raises questions about the national vaccine program.
Microbiologists from the University of NSW have found variants of the pertussis bacteria with a particular genetic signature have increased to 86 per cent of all samples taken from infected people after a continuing disease epidemic began in 2008.
Although the strains were present in Australia as early as 2000, they accounted for only 31 per cent of all samples collected between 2000 and 2007 — suggesting they have flourished alongside the current vaccine.
An acellular vaccine — introduced in Australia in 1997 after concerns about side-effects from the previous whole cell version — appeared to have promoted the spread of these variants, Dr Lan said, which overseas authorities had linked to “higher virulence on the basis of hospitalisation and case mortality data”.
He warned that other countries using similar vaccines should be alert for shifts in genetic features detected in the whooping cough bug.
Hepatitis B Virus
In southern Italy, 44 contacts of hepatitis B virus carriers, including infants of carrier mothers, became HBsAg positive despite passive and active immunisation according to standard protocols. In 32 of these vaccinees infection was confirmed by the presence of additional markers of viral replication.
The circulation of HBV encoding envelope mutations selected by antiviral agents requires further investigation to determine whether they may be transmitted and therefore represent a public health concern. This issue may be of particular relevance in populations where genotype A is predominant.
HBV strains containing vaccine escape mutation patterns are replication competent and are shown to be infectious and pathogenic.
When the live-virus version of the vaccine, called Oral Polio Vaccine (OPV) evolves, he says, it can act like wild poliovirus and continue the threat of contagion. Over time, the vaccine can mutate, and even a 1 percent genomic change in the virus permits the virus to behave like a wild poliovirus. As a result, they are finding evidence of vaccine-derived polio cases in humans.
Gene Swapping – Lethal Viruses from Vaccines
Three vaccines used to prevent respiratory disease in chickens have swapped genes, producing two lethal new strains that have killed tens of thousands of fowl across two states in Australia
The creation of the deadly new variant was only possible because the vaccines contained live viruses, even though they were weakened forms, said Joanne Devlin, lead author of the paper published in the journal Science.
Scientists from a drug company and two labs analyzed more than 21,000 bacterial samples from around the nation and found that one type of resistant strain is increasing in ear infections.
University of Iowa researchers found the strain accounted for 35 percent of penicillin-resistant infections in 2004-05, compared with less than 2 percent the year before the new vaccine came out.